Search results for " Anticardiolipin"

showing 5 items of 5 documents

Thrombotic events in systemic lupus erythematosus. Its association with acquired and inherited thrombophilic defects.

2008

Systemic lupus erythematosus (SLE) is characterised by increased venous and arterial thrombotic risk. Although antiphospholipid antibodies (APAs) have been shown to be related with thrombotic tendency in these patients, in more than 40% of them, thrombosis occurs without the presence of such antibodies. We analysed the association of venous and arterial thrombotic events with acquired (anticardiolipin antibodies (ACAs) and lupus anticoagulant (LA)) and inherited (antithrombin (AT), protein C (PC), protein S (PS) deficiencies, factor V Leiden and the prothrombin G20210A mutation), thrombophilic risk factors in 86 SLE patients and 89 healthy controls. Patients showed a higher significant perc…

AdultMalemedicine.medical_specialtyPhysiologyGastroenterologyProtein SRisk FactorsPhysiology (medical)Internal medicinemedicineFactor V LeidenHumansLupus Erythematosus SystemicRisk factorLupus anticoagulantbiologybusiness.industryAntithrombinThrombosisHematologyBlood ProteinsMiddle Agedmedicine.diseaseThrombosisVenous thrombosisAntibodies AnticardiolipinLupus Coagulation InhibitorImmunologyMutationbiology.proteinFemaleCardiology and Cardiovascular MedicinebusinessProtein Cmedicine.drugClinical hemorheology and microcirculation
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Antiphospholipid syndrome in obstetrics.

2003

Antiphospholipid syndrome (APLS) in pregnancy is characterized by the presence of autoantibodies in association with recurrent fetal loss and severe complications such as preeclampsia, fetal growth retardation, or placental insufficiency. The most clinically important serologic markers are lupus anticoagulant, anticardiolipin antibodies, and recently anti-beta-2-glycoprotein 1 antibodies. At present, standardization does not exist and a definitive association between specific clinical manifestation and antibody level is not yet known. Experimental data gave evidence that passive transfer of antiphospholipid antibodies result in clinical manifestation of APLS, that is, fetal loss and thromb…

Adultmedicine.medical_specialtyAbortion HabitualPlacental insufficiency030204 cardiovascular system & hematologyGastroenterologyPreeclampsia03 medical and health sciences0302 clinical medicinePre-Eclampsiaimmune system diseasesAntiphospholipid syndromePregnancyInternal medicinemedicinePrevalenceHumansThrombophiliaAnnexin A5030203 arthritis & rheumatologyLupus anticoagulantAspirinPregnancyFetal Growth RetardationAspirinbusiness.industryHeparinStandard treatmentInfant NewbornAnticoagulantsImmunoglobulins IntravenousHematologyGeneral MedicineHeparinmedicine.diseaseAntiphospholipid SyndromePlacental InsufficiencyThrombocytopeniaAbortion SpontaneousPregnancy ComplicationsAntibodies AnticardiolipinLupus Coagulation InhibitorImmunologyPrednisoneFemalebusinessImmunity Maternally-AcquiredImmunosuppressive Agentsmedicine.drugClinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis
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Immunological pattern in patients with 21-hydroxylase deficiency.

1994

The aim of this work was to perform an immunological study in six patients with 21 hydroxylase deficiency in mild form (M210HD) and in 2 patients with 21-hydroxylase deficiency in classical form (C210HD) and in their parents, in whom a previous HLA,C4,Bf typing demonstrated high prevalence of DR5 and phenotypic absence of fraction C4B of complement (C4BQO). This study contains the evaluation of C3, IgA, IgG, IgM levels, anticardiolipin antibodies (IgG and IgM) and circulating immunocomplexes. A study of lymphocyte subsets was also performed. Among M210HD 1 patient showed presence of anticardiolipin antibodies both IgM and IgG; this patient had shown antinuclear antibodies in a previous stud…

CD4-Positive T-LymphocytesMalemedicine.medical_specialtyAnti-nuclear antibodyAdolescentEndocrinology Diabetes and MetabolismHuman leukocyte antigenImmunogeneticsBiologymedicine.disease_causeT-Lymphocytes RegulatoryAutoimmunityPathogenesisEndocrinologyImmune systemImmunityInternal medicinemedicineHumansFamily HealthAdrenal Hyperplasia CongenitalImmunityT-Lymphocytes Helper-InducerEndocrinologyImmunoglobulin MAntibodies AnticardiolipinImmunoglobulin GImmunologybiology.proteinFemaleAntibodyJournal of endocrinological investigation
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Anticardiolipin antibodies in patients with liver disease.

1999

OBJECTIVE: Our aim was to test the hypothesis that anticardiolipin antibodies (aCL) may cause an antiphospholipid syndrome and thrombotic events in patients with liver disease. METHODS: aCL were measured in 116 healthy controls and 372 patients with liver disease of different stage and etiology: 136 cases secondary to hepatitis C virus (HCV) infection, 139 due to hepatitis B virus (HBV) infection, 69 with alcoholic liver damage, and 28 cryptogenic in origin. Prior thrombotic events were recorded. The results were related to age, gender, stage, severity, and etiology of the liver disease, as well as to the occurrence of organ- and nonorgan-specific autoantibodies. RESULTS: aCL were positive …

Liver CirrhosisMalemedicine.medical_specialtyAlcoholic liver diseasemedicine.disease_causeGastroenterologyLiver diseaseHepatitis B ChronicAntiphospholipid syndromeInternal medicinemedicineHumansAutoantibodiesHepatitis B virusHepatologybusiness.industryLiver DiseasesGastroenterologyMuscle SmoothThrombosisOdds ratioHepatitis CHepatitis C ChronicMiddle AgedHepatitis BAntiphospholipid Syndromemedicine.diseaseLogistic ModelsAntibodies AnticardiolipinHepatocellular carcinomaChronic DiseaseImmunologyFemalebusiness
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Effects of anti-cardiolipin antibodies and IVIg on annexin A5 binding to endothelial cells: implications for cardiovascular disease

2010

Anti-phospholipid antibodies (aPL), including anti-cardiolipin antibodies (aCL), are risk factors for cardiovascular disease (CVD) in the general population and in patients with the anti-phospholipid syndrome (APS; Hughes syndrome). APS may be primary but is also common in patients with systemic lupus erythematosus (SLE). The anti-coagulant protein annexin A5 (ANXA5) is implicated in CVD by interfering with phospholipids and aPL.ANXA5 binding to human umbilical venous endothelial cells (HUVECs) was determined by flow cytometry.When cells were cultured in serum from APS patients with a high aPL titre (aPL-S), binding of ANXA5 to HUVECs was reduced. Monoclonal immunoglobulin (Ig)G aPL against…

MaleUmbilical Veinsmedicine.medical_specialtyEndotheliumPopulationImmunologyImmunoglobulin Gchemistry.chemical_compoundRheumatologyReference Valuesimmune system diseasesAntiphospholipid syndromeInternal medicineCardiolipinmedicineHumansImmunology and AllergyAnnexin A5educationneoplasmsCells CulturedProbabilityeducation.field_of_studyBinding Sitesbiologybusiness.industryEndothelial CellsImmunoglobulins IntravenousGeneral MedicineAntiphospholipid SyndromeFlow Cytometrymedicine.diseaseEndocrinologymedicine.anatomical_structurechemistryCardiovascular DiseasesAntibodies AnticardiolipinCase-Control StudiesImmunoglobulin Gbiology.proteinFemaleAnti-cardiolipin antibodiesAntibodyAnnexin A5businessScandinavian Journal of Rheumatology
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